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First, we would know the absolutelocal O 2 requirements for the most vulnerable tis-sues in any critically ill patients, enabling the cli-nician to decide on whether maximal oxygenationwas necessary, or whether a degree (and to whatextent) permissive hypoxemia might be titrated.Second, we would understand how ventilationinjures lungs (and worsens outcome), and for any“necessary” burden of mechanical ventilation(that has been optimized to minimally injure),we could treat a patient with a speci? c pathwayinhibitor or activator that would bypass the bio-chemical mechanism whereby the ventilator wascausing residual injury. When the person isin a similar environmental, situational, or emotional situation, the brainwill crack open the capsule. 2009 ).Inhaled iloprost was ?rst used in patients withacute respiratory failure and increased pulmo-nary arterial pressure accompanied by distur-bances of gas exchange and then in acute heartright failure, and later effects were compared toinhaled nitric oxide (Zwissler et al. This trial was performedin the pre- surfactant era, limiting interpretationof results (HiFO Study Group 1993; Bhuta andHenderson- Smart 2000)

This trial was performedin the pre- surfactant era, limiting interpretationof results (HiFO Study Group 1993; Bhuta andHenderson- Smart 2000). re-ported that he had chemotherapy and radiation and had been monitored for 2 years

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